Well I do not have much time to post this week but wanted to do a quick fooooortay on a few things I have noticed…

Before I got stuck in my eating disorder I was this very charismatic, outgoing, unpredictable person. There are a few things lately which lead me to believ I am coming back to this person

  • I suffer some real ADD that makes me laugh so hard with my friends about nothing
  • I love Jersey Shore; I sit through the whole thing…GUIDOOOO. I need to get a GTL bag
  • I got a hold of camouflage duct tape. I put it on my hand and went up to my dad (visited my parents) who wears camo pants 24/7 and was like “look you cant see me hand!!”….I’m such a retard haha
  • I have been talking on the phone ten times more than I use to. Good things to come J
  • I have more energy than I know what to do with. I feel like someone spiked my body with a cross of Ritalin and speed. I cannot slow down!


And some good reading I have come across this week:

All from Stephanie’s Blog, amazing information here

http://stephanie-on-health.blogspot.com/ :

It has been estimated that 70% of America’s children are currently deficient in vitamin D [20] (Details) . This is not surprising, given current medical advice. The sunscreen industry lobby has convinced most Americans, including medical experts, that the sun should be aggressively avoided to prevent skin cancer. This, in spite of the fact that the sun is an excellent source of vitamin D, allowing the skin to manufacture it directly from cholesterol. Moreover, vitamin D is protective against all cancers (Details) a characteristic which, in my view, more than compensates for any extra skin cancer risk incurred by sunbathing. Vitamin D deficiency is also associated with an increased risk of high blood pressure and diabetes [35]. In order to get vitamin D from food, it is necessary to eat animal fats; animals manufacture vitamin D, a fat-soluble vitamin, and store it in their fat cells.

The American medical establishment is heavily entrenched in the idea that dietary fat is unhealthy. People are encouraged to adopt low fat diets, which inevitably lead to an increase in their intake of carbs and sugars, as much of the fat removed in foods is replaced with sugars to make them palatable. Many foods are also often highly processed and easily digested, leading to a rapid rise in blood sugar. At the same time, foods containing vitamin D are avoided, due to their universally high fat content.

Vitamin D is crucial to the absorption of calcium from the gut into the blood stream, and both vitamin D and calcium are important catalysts in crucial biological processes. Fats also promote the uptake of calcium in the gut, whereas dietary fiber, touted as being healthy, impedes it [38] (Details) . These three nutrients, fats, vitamin D, and calcium, have intricate mutual dependencies that make it important to consume them together. Americans are deficient in these important nutrients because of their perceived need to pursue a low fat diet and avoid sun exposure.

ADHD and Anorexia:

Fat cells are part of the endocrine system, and, as I’ve discussed before, they have the power to influence the degree to which muscle cells prefer glucose versus fats as an energy source. They exercise this control by releasing two signaling peptides: leptin and adiponectin. Adiponectin promotes glucose consumption by the muscles, and it also acts directly on the fat cells to encourage them to take up glucose and convert it to fat. Leptin, on the other hand, stimulates the muscles to prefer fat consumption over glucose consumption.

Statistically, children with ADHD have an abnormally efficient glucose metabolism rate, i.e., for the same amount of insulin, blood sugar levels drop more quickly after a meal than in other children. This observation suggests that their fat cells have arranged a set point of a high adiponectin to leptin ratio, such that the muscles prefer glucose over fats, and fat cells are predisposed to convert glucose to fat. The glucose levels drop more quickly because the muscles and fat cells are using more of it.

Anorexics, children who intentionally starve themselves, are known to have extremely efficient glucose metabolism (tend towards hypoglycemia) and also to have a high ratio of adiponectin to leptin concentrations . This strategy maximizes availability of fatty acids to the heart and brain. It is curious that anorexia is much more common in girls, and ADHD is much more common in boys.

Researchers at Harvard Medical School suspected that there might be an association between anorexia and ADHD. To test this hypothesis, they compared girls with ADHD against a control group to see whether the ones with ADHD were predisposed towards anorexia(ADHD and Anorexia). The results showed that girls with ADHD were 3.6 times more likely than the control group to develop an eating disorder. I have come to believe that anorexia is a technique to combat ADHD that girls are able to adopt, whereas boys do not have enough fat cells to carry out the task of converting glucose to fat. Ritalin is well known to reduce appetite, and long term use can lead to an anorexia-like condition. It may well work, in part, because it achieves this ultra-thin state, thus conserving fats by minimizing the consumption of fat by cells that can get by on glucose.

TWO biggest problems in health:

For several decades now, Americans have come to believe that the following two practices are foundational in a healthy lifestyle:  eat a low-fat diet, and  stay away from the sun. Additionally, if people consume adequate amounts of calcium, then all three nutritional deficiencies that have led to obesity will be overcome: vitamin D, calcium, and dietary fat.

The lack of adequate dietary fat contributes to the metabolic syndrome in at least four ways:  vitamin D is only available in fatty food sources because it is a fat-soluble vitamin, calcium uptake is more efficient when the calcium is consumed with dietary fats, calcium uptake depends critically on the presence of vitamin D, which is deficient due to (1) above, and the burden of fat cells to manufacture fatty acids from sugar is alleviated by the dietary availability of fats from ingested food sources.

I would also argue that one should make sure to ingest adequate amounts of dietary fat, especially dairy fat . Whole milk is particularly outstanding because it contains substantial amounts of calcium and vitamin D, and it contains the necessary fat to assure that these two elements will be well utilized rather than just passing through the digestive system unabsorbed. Animal fats such as bacon are good sources of vitamin D, while also supplying fatty acids to help with energy needs. Fatty fish such as salmon and sardines are particularly good because they contain both omega-3 fats and vitamin D. One should assiduously avoid the trans fats found in processed foods such as cookies, crackers, and margarine. Butter and eggs are also healthy choices. Egg yolk is particularly good because it contains both fats and vitamin D. Nuts, particularly walnuts, almonds, and macademia nuts, are excellent sources of omega 3 fats.

The fat cells are able to influence the muscles to preferentially take up fats rather than glucose by releasing certain hormones into the blood, hormones that also have a powerful influence over appetite. One of these hormones is leptin. While leptin influences the muscle cells indirectly through its signaling in the hypothalamus, it also stimulates the muscle cells directly, and influences them to oxidize fatty acids in their mitochondria. Leptin also encourages the fat cells to release their fats through lipolysis. All of these actions work in concert to redirect fuel usage away from glucose. The programming of the muscles to preferentially consume fats aligns well with the fat cells’ infusion of fats into the blood and absorption of sugars through their fat-producing factories.

Leptin also has the effect, via the hypothalamus and pituitary gland, of suppressing appetite. Adiponectin is another hormone released by fat cells, and it is generally agreed that adiponectin induces hunger. Leptin and adiponectin levels would ordinarily fluctuate throughout the day, with leptin levels rising at night to encourage a switch from glucose-based to fat-based energy management. However, in the obese person, the leptin levels are typically high all the time, and the adiponectin levels are kept very low. High levels of leptin in the blood signal to the appetite center in the brain a sense of being full, whereas high levels of adiponectin are hunger-inducing. This means that the obese are being informed both that they are full, and that they are not hungry. You would think that this would protect them from overeating. However, it is likely that the observed insensitivity to leptin as an appetite suppressant in the obese is also related to calcium depletion, because the signaling mechanisms that respond to leptin in both the hypothalamus (Details) and the pituitary gland (Details) depend on changes in internal calcium concentrations

The result of these three deficiencies is defective glucose uptake in both muscle and fat cells. The obese person becomes trapped in an endless metabolic cycle of trying to supply the energy needed for a steadily increasing demand. The fat cells are at the center of the storm, because they are burdened with the arduous assignment of converting the excess consumed sugars and carbohydrates into fat. The fat cells must do this because the muscle cells are impaired with a malfunctioning ability to metabolise sugars. Even if the metabolic problem were not fixed, if the obese person simply ate more fat, and therefore consumed fewer carbs, the fat cells’ burden would be greatly alleviated. In addition, getting plenty of vitamin D and calcium, either through diet or sun exposure, would alleviate the core problem of impaired glucose transport across the cell wall. Now that the heart and muscles can utilize sugars directly, the excessive burden on the fat cells to expand and proliferate is relieved, and the body fat will inevitably melt away.

The metabolic syndrome is a term used to encapsulate a complex set of markers associated with increased risk to heart disease. The profile includes insulin resistance and dysfunctional glucose metabolism in muscle cells, excess triglycerides in the blood serum, high levels of LDL, particularly small dense LDL, the worst kind  low levels of HDL (the “good” cholesterol) and reduced cholesterol content within the individual HDL particles, elevated blood pressure, and obesity, particularly excess abdominal fat. I have argued previously that this syndrome is brought on by a diet that is high in empty carbohydrates (particularly fructose) and low in fats and cholesterol, along with a poor vitamin D status [Seneff2010]. While I still believe that all of these factors are contributory, I would now add another factor as well: insufficient dietary sulfate.

  • A recent analysis of data from the Nurses’ Health Study, an ambitious long-term study involving over 18,000 nurses, showed that fat in dairy consumption was associated with high fertility. Women who said they ate low-fat diary increased their risk of infertility by 85%, whereas women who consistently ate high-fat dairy decreased their risk by 27%. Fertility is an indicator of the degree to which the body perceives that it is prepared to support a fetus. Breast milk has an extremely high fat content, significantly higher than that of cow’s milk. It then seems logical that, once mother’s milk is replaced with table foods, these foods should continue to be high in fat content.

Heres an interesting tidbit on ketosis and pregnancy: